| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 2044392 | Current Biology | 2006 | 4 Pages |
Abstract
DNA polymerase zeta and Rev1 play key roles in replication past DNA lesions. New work shows that the yeast checkpoint kinase Mec1 recruits a complex consisting of polymerase zeta and Rev1 to DNA double-strand breaks. This study highlights the role of polymerases that mediate translesion synthesis in the response to DNA double-strand breaks.
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Authors
Nadine K. Kolas, Daniel Durocher,