Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
2836581 | Physiological and Molecular Plant Pathology | 2011 | 6 Pages |
The present study investigated resistance against Botrytis cinerea after heat shock treatment in melon plants. Heat shock at 50 °C for 20 s 0–24 h before inoculation resulted in maximal B. cinerea symptom reduction and peroxidase gene expression, which peaked 12 and 72 h post-treatment and decreased 24–48 h post-treatment, suggesting pathogenesis-related protein expression priming. Hot water dipping did not directly inhibit mycelia growth. Plants treated with 2-benzisothiazol-3(2H)-one 1,1-dioxide, which induces systemic acquired resistance, demonstrated higher peroxidase gene expression but no B. cinerea resistance, indicating possible involvement of additional novel mechanisms in heat shock-activated resistance of melon against B. cinerea.
► BIT did not reduce gray mold disease symptoms. ►SAR mediate partly the protective effect of heat shock treatment. ► Mechanism may be involved in the disease resistance induced by heat shock against B. cinerea.