| Article ID | Journal | Published Year | Pages | File Type | 
|---|---|---|---|---|
| 3311536 | Hepatology Research | 2006 | 8 Pages | 
Abstract
												The results suggest that carcinogen metabolism and DNA-repair pathways may simultaneously modulate the risk of HCC for Guangxi population, and, particularly for these having high-level or long-term AFB1 exposure.
											Keywords
												
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											Authors
												Xi Dai Long, Yun Ma, Yi Ping Wei, Zhou Lin Deng, 
											