| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9000632 | Experimental and Toxicologic Pathology | 2005 | 8 Pages |
Abstract
These results demonstrated that indomethacin-induced gastric mucosal damage was accompanied by the development of oxidative stress, evidenced by the accumulation of MDA. AMFJ-pretreatment decreased the gastric lesions caused by indomethacin. It could be suggested that this effect of AMFJ was probably due to the increased mucus production and interference with oxidative stress development as evidenced by the decreased plasma and gastric mucosal MDA.
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Authors
S. Valcheva-Kuzmanova, K. Marazova, I. Krasnaliev, B. Galunska, P. Borisova, A. Belcheva,