Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
9147547 | Journal of Insect Physiology | 2005 | 8 Pages |
Abstract
Host hemocytes stained with FITC-labeled phalloidin, which binds to filamentous actin, were used to observe hemocyte disruption in parasitized and virus-injected hosts and a comparison was made to hemocytes of nonparasitized control larvae. At 24Â h post-parasitization host hemocytes were significantly altered compared to those of nonparasitized larvae. Hemocytes from newly parasitized hosts displayed blebbing, inhibition of spreading and adhesion, and overall cell disruption. A CrV1-homolog gene product was localized in host hemocytes using polyclonal CrV1 antibodies, suggesting that CrV1-like gene products of C. congregata's bracovirus are responsible for the impaired immune response of the host.
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Authors
Kevin E. Amaya, Sassan Asgari, Richard Jung, Melissa Hongskula, Nancy E. Beckage,