| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5851402 | Food and Chemical Toxicology | 2013 | 8 Pages |
Abstract
⺠H. pylori induced NF-κB activation in the H. pylori-infected cells. ⺠Cellular oxidative burst occurs. ⺠Apigenin treatment modulates NF-κB, ROS, and MUC-2 expressions. ⺠Apigenin plays a key role in anti-H. pylori-induced cell inflammation.
Keywords
NF-κBH2DCF-DAT4SScagAextracellular signal regulated kinase 1/2NIKVacAIKKα/βIL-8TRL4dihydrodichlorofluorescein diacetateEtBrAP-1MUC-2PGE2iNOSLPSICAM-1JnkCOX-2NF-κB-inducing kinaseERK1/2MAPKNOxROSApigeninethidium bromideinflammationNADPH oxidaseInterleukin-8dihydroethidiumGastric cancerinducible nitric oxide synthaseVacuolating cytotoxinCyclooxygenase-2nuclear factor kappa Blipopolysaccharideintercellular adhesion molecule-1mucin-2Helicobacter pyloriactivator protein 1mitogen-activated protein kinaseProstaglandin E2Oxidative burstReactive oxygen speciesToll-like receptor 4
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Authors
Yuan-Chuen Wang, Kai-Ming Huang,