| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 5851518 | Food and Chemical Toxicology | 2013 | 10 Pages |
Abstract
⺠Res protects DRG neurons from EtOH-induced cell apoptosis. ⺠Res improves EtOH-induced reduction of neurite outgrowth and cell migration. ⺠Res alleviates EtOH-induced oxidative/nitrosative stress in DRG neurons. ⺠The protective effects of Res are associated with the activation of AMPK and SIRT1.
Keywords
WST-1PGC-1αMAP2GSK3βNGFRESDAPIRNSsirtuin 1GSHNCAMDASirt1LKB1PBSDRGPARP-1AMPKdorsal root ganglion4′,6-diamidino-2-phenylindoleROSEthanolcompound COxidative stressParApoptosisResveratrolembryonic daySODserine/threonine kinase 11Superoxide dismutasenerve growth factorPhosphate buffered salinemalondialdehydeNicotinamidemicrotubule-associated protein 2Poly(ADP-ribose) polymerase-1dorsal root gangliaGlutathioneGlycogen synthase kinase 3βreactive nitrogen speciesReactive oxygen species
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Authors
Hongtu Yuan, Weiwei Zhang, Hao Li, Cheng Chen, Huaxiang Liu, Zhenzhong Li,