Increase of delayed rectifier potassium currents in large aspiny neurons in the neostriatum following transient forebrain ischemia
Keywords: ایسکمی مغزی; striatum; cerebral ischemia; neuronal death; interneuron; membrane excitability; potassium channel; ACSF; artificial cerebrospinal fluid; H-89; N-[2-((p-bromocinnamyl)amino)ethyl]-5-isoquinolinesulfonamide; Ik; delayed rectifier potassium current; ISI; in