Endoplasmic reticulum stress-mediated inhibition of NSMase2 elevates plasma membrane cholesterol and attenuates NO production in endothelial cells
Keywords: NO; nitric oxide; ER; endoplasmic reticulum; BAEC; bovine aortic endothelial cells; eNOS; endothelial nitric oxide synthase; NSMase or NSM; neutral sphingomyelinase; SREBP-2; sterol-responsive element-binding protein; Tm; tunicamycin; Pm; palmitate; RNAi;