Antiallergic drugs, azelastine hydrochloride and epinastine hydrochloride, inhibit ongoing IgE secretion of rat IgE-producing hybridoma FE-3 cells
Keywords: IgE; Antiallergic drug; Nuclear factor-kappa B (NF-κB); I kappa B alpha (IκBα); Activation-induced cytidine deaminase (AID)