
Keywords: CCl4-induced hepatotoxicity; Ginsenoside Rg1; InflammationAMPK, AMP-activated protein kinase; Rg1, Ginsenoside Rg1; MPO, myeloperoxidase; MDA, malondialdehyde; NF-κB, Nuclear factor-kappaB; TNF-α, tumor necrosis factor-α; IL-6, interleukin-6; iNOS, nitric