
Recovery of redox homeostasis altered by CuNPs in H4IIE liver cells does not reduce the cytotoxic effects of these NPs: An investigation using aryl hydrocarbon receptor (AhR) dependent antioxidant activity
Keywords: CuNP; copper nanoparticle; ROS; reactive oxygen species; AhR; aryl hydrocarbon receptor; ÃNF; Ã-Naphthoflavone; αNF; α-Naphthoflavone; GST; glutathione-S-transferase; GSH; glutathione; GSSG; oxidised glutathione; Nrf2; nuclear factor erythroid-2-relat