Tenofovir alafenamide (TAF) does not deplete mitochondrial DNA in human T-cell lines at intracellular concentrations exceeding clinically relevant drug exposures
Keywords: Mitochondrial toxicity; mtDNA synthesis; T-cells; NRTI; TAF; Tenofovir alafenamide; d4T; stavudine; ddC; dideoxycytidine; ddI; dideoxyinosine; NRTI; nucleoside reverse transcriptase inhibitor; PBMC; peripheral blood mononuclear cell; TAF; tenofovir alafen