Dysregulation of synaptic proteins, dendritic spine abnormalities and pathological plasticity of synapses as experience-dependent mediators of cognitive and psychiatric symptoms in Huntington’s disease
Keywords: BDNF, brain-derived neurotrophic factor; CAG, cytosine-adenine-guanine trinucleotide; EE, environmental enrichment; F-actin, actin filaments; HD, Huntington’s disease; htt, huntingtin; LTD, long-term depression; LTP, long-term potentiation; PSD, postsynap