
Doxorubicin induced myocardial injury is exacerbated following ischaemic stress via opening of the mitochondrial permeability transition pore
Keywords: FCCP، کربنیل سیانید p- (trifluoromethoxy) phenylhydrazone; ANT, Adenine nucleotide translocase; ATP, Adenosine triphosphate; CsA, Cyclosporin A; Cyp-D, Cyclophilin D; Dox, Doxorubicin; Erk, Extracellular signal-regulated kinase; FCCP, Carbonyl cyanide p-(trifluoromethoxy)phenylhydrazone; HR, Hypoxia reoxygenation