Lack of CAK complex accumulation at DNA damage sites in XP-B and XP-B/CS fibroblasts reveals differential regulation of CAK anchoring to core TFIIH by XPB and XPD helicases during nucleotide excision repair
Keywords: TFIIH, transcription factor II H; CAK, Cdk-activating kinase complex; UV, ultraviolet light; 6-4PP, 6-4 pyrimidine-pyrimidone photoproduct; CPD, cyclobutane pyridine dimer; NER, nucleotide excision repair; GGR, global genomic repair; TCR, transcription-co