
Keywords: AML; acute myeloid leukemia; ChIP; Chromatin immunoprecipitation; CML; chronic myeloid leukemia; ECM; extracellular matrix; GO; gene ontology; CD29/ITGB1; integrin beta 1; HIP1; huntingtin interacting protein; DAB2; disabled homolog 2; SDC1; syndecan 1; L