Hepatitis B virus X protein increases the IL-1β-induced NF-κB activation via interaction with evolutionarily conserved signaling intermediate in Toll pathways (ECSIT)
Keywords: AP-1, activating protein-1; ECSIT, evolutionarily conserved signaling intermediate in Toll pathways; HB, xhepatitis B virus X protein; I, κBinhibitor of NF-κB; IRAK, interleukin-1 receptor-associated kinase; JNK, c-Jun N-terminal kinase; MAPK, mitogen-act