Acute nitrogen dioxide (NO2) exposure enhances airway inflammation via modulating Th1/Th2 differentiation and activating JAK-STAT pathway
Keywords: ژن موچین; NO2; nitrogen dioxide; MUC5AC; mucin gene; IL-1β; Interleukin-1β; IL-6; Interleukin-6; ICAM-1; intercellular adhesion molecule; Th2; subtype of helper T lymphocyte; JAK; Janus kinase; STAT; signal transducer and activator of transcription; IL-4; Interle