
The NADPH oxidase inhibitor DPI can abolish hypoxia-induced apoptosis of human kidney proximal tubular epithelial cells through Bcl2 up-regulation via ERK activation without ROS reduction
Keywords: Renal ischemia–reperfusion injury; Human renal proximal tubular epithelial cells; Reactive oxygen species; Cell viability; Apoptosis; Antioxidant; NADPH oxidase inhibitor