Upregulation of nitric oxide synthase II contributes to apoptotic cell death in the hippocampal CA3 subfield via a cytochrome c/caspase-3 signaling cascade following induction of experimental temporal lobe status epilepticus in the rat
Keywords: Experimental temporal lobe status epilepticus; Apoptosis; Nitric oxide synthase II; Cytochrome c; Caspase-3; Hippocampal CA3 subfield