Arsenic trioxide induces procoagulant activity through phosphatidylserine exposure and microparticle generation in endothelial cells
Keywords: As2O3, arsenic trioxide; APL, acute promyelocytic leukemia; EMPs, endothelial microparticles; HUVECs, human umbilical vein endothelial cells; PCA, procoagulant activity; PFP, particle-free plasma; PML-RARα, promyelocytic leukemia-retinoic acid receptor-α;