
The effect of pharmacological PI3Kγ inhibitor on eotaxin-induced human eosinophil functions
Keywords: Asthma; Eosinophil; Eotaxin; PI3KγAHR, airway hyperresponsiveness; ERK, extracellular signal-regulated kinase; GPCR, G protein coupled receptor; PI3K, phosphoinositide-3 kinase; RPMI, Roswell Park Memorial Institute; PI, propidium iodine