
Pirfenidone attenuates IL-1β-induced COX-2 and PGE2 production in orbital fibroblasts through suppression of NF-κB activity
Keywords: پیرفنیدون; thyroid-associated ophthalmopathy; orbital fibroblast; pirfenidone; COX-2; PGE2; IL-1β; NF-κB; COX; cyclooxygenase; IL; interleukin; NF; nuclear factor; PG; prostaglandin; TAO; thyroid-associated ophthalmopathy;