Activation of ATP-sensitive potassium channels prevents the cleavage of cytosolic μ-calpain and abrogates the elevation of nuclear c-Fos and c-Jun expressions after hypoxic-ischemia in neonatal rat brain
Keywords: فیزیولوژی کانال پالادیوم، فارماکولوژی و مدولاسیون; Excitable membranes and synaptic transmission; Potassium channel physiology, pharmacology, and modulation; Mitochondrial ATP-sensitive K+ channel; μ-Calpain; Hypoxic-ischemic brain injury; c-Fos; c-Jun;