Oxidative stress alters base excision repair pathway and increases apoptotic response in apurinic/apyrimidinic endonuclease 1/redox factor-1 haploinsufficient mice
Keywords: فعالیت های بازسازی; Apurinic/apyrimidinic endonuclease 1/redox factor-1; Redox activity; Base excision repair; Oxidative DNA damage; NF-κB; Apoptosis; Liver; Free radicals