
Escape of intracellular Shigella from autophagy requires binding to cholesterol through the type III effector, IcsB
Keywords: Shigella; IcsB; Autophagy; Cholesterol binding; T3SA; type III secretion apparatus; CBD; cholesterol binding domain; SBD; sterol binding-like domain; RID; Rho GTPase inactivation domain; MDAMC; human breast carcinoma epithelial cells; MβCD; methyl-beta-c