uPA and uPAR shRNA inhibit angiogenesis via enhanced secretion of SVEGFR1 independent of GM-CSF but dependent on TIMP-1 in endothelial and glioblastoma cells
Keywords: uPA and uPAR; shRNA; Angiogenesis; GlioblastomauPA, urokinase plasminogen activator; uPAR, urokinase plasminogen activator receptor; GM-CSF, granulocyte macrophage colony stimulating factor; SVEGFR1, soluble vascular endothelial growth factor receptor 1;