IL-36γ induces a transient HSV-2 resistant environment that protects against genital disease and pathogenesis
Keywords: سلول های اپیتلیال واژن; Vaginal epithelial cells; Chemokines; Antimicrobial peptides; Polymorphonuclear leukocytes; 3-D bioreactor model; HSV-2 mouse model; IL-36 family members; Host defense; Sexually transmitted infections;