A peptide derived from the Wiskott-Aldrich syndrome (WAS) protein-interacting protein (WIP) restores WAS protein level and actin cytoskeleton reorganization in lymphocytes from patients with WAS mutations that disrupt WIP binding
Keywords: Wiskott-Aldrich syndrome; WASP; WIP; T cells; actin cytoskeleton; aa; Amino acid; EGFP; Enhanced green fluorescent protein; EBV-B; EBV-transformed B; Ena/VASP; Enabled/vasodilator-stimulated phosphoprotein; EVH1; Ena-VASP homology domain 1; MFI; Mean fluo