Amyloid toxicity is enhanced after pharmacological or genetic invalidation of the Ï1 receptor
Keywords: σ1 گیرنده; AD; Alzheimer's disease; ALS; amyotrophic lateral sclerosis; APP; amyloid precursor protein; Aβ25-35; amyloid-β[25-35] peptide; BDNF; brain-derived neurotrophic factor; BiP; 78 kDa glucose-regulated protein; cAMP; cyclic adenosine monophosphate; CREB;