
Acute nicotine treatment attenuates lipopolysaccharide-induced cognitive dysfunction by increasing BDNF expression and inhibiting neuroinflammation in the rat hippocampus
Keywords: فاکتور نوروئرتفیک مغزی (BDNF); Acute nicotine treatment; Lipopolysaccharide (LPS); Cognitive dysfunction; Neuroinflammation; Brain-derived neurotrophic factor (BDNF);