
H2S protects lipopolysaccharide-induced inflammation by blocking NFκB transactivation in endothelial cells
Keywords: H2S; Cystathionine gamma-lyase; LPS; Endothelial cells; Inflammation; NFκB; ChIP; chromatin immunoprecipitation; CRISPR; clustered regulatory interspersed short palindromic repeats; CSE; Cystathionine gamma-lyase; H2S; Hydrogen sulfide; HUVEC; Human umbi