Keywords: نوشیدن در تاریکی; Alcohol; Maternal separation; Adolescence; Endocannabinoids; Monoamines; Striatum; 2-AG; 2-arachidonoylglycerol; 2-LG; 2-linoleoylglycerol; 2-OG; 2-oleoylglycerol; 5-HIAA; 5-hydroxyindoleacetic acid; AEA; N-arachidonoylethanolamine; BAC; Blood Alcohol Con
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Keywords: نوشیدن در تاریکی; Ethanol; Kynurenine; Kynurenic acid; KMO; Ro 61-8048; Binge drinking; Drinking in the dark; Ethanol (PubChem CID: 702); Ro 61-8048 (PubChem CID: 5282337); L-kynurenine (PubChem CID: 161166); CH-223191 (PubChem CID: 3091786); 6,2â²,4â²-Trimethoxyflavone
Keywords: نوشیدن در تاریکی; (2-AG); 2-arachidonoyl-glycerol; (â9-THC); delta-9-tetrahydrocannabinol; (AA); Alko Alcohol preferring; (ABHD); alpha/beta hydrolase domain-containing protein; (ADE); alcohol deprivation effect; (AEA); N-arachidonoylethanolamine; (CB1); cannabinoid rece
Keywords: نوشیدن در تاریکی; Binge; Drinking in the dark; Ethanol consumption; Genetics; Selective breeding
Keywords: نوشیدن در تاریکی; Adolescence; DBA/2J; C5BL/6J; Ethanol; Motor impairment; Drinking in the dark; Intermittent access; Mouse; Alcohol; Sex differences;
Keywords: نوشیدن در تاریکی; Ethanol binge drinking; Conditioned fear; Selective breeding; Drinking in the dark; Mouse; Genetics
High Drinking in the Dark (HDID) mice are sensitive to the effects of some clinically relevant drugs to reduce binge-like drinking
Keywords: نوشیدن در تاریکی; Genetic animal model; Drinking in the dark; Binge; Pharmacotherapy; Mouse;
Archival ReportExtended Amygdala to Ventral Tegmental Area Corticotropin-Releasing Factor Circuit Controls Binge Ethanol Intake
Keywords: نوشیدن در تاریکی; Binge drinking; Corticotropin-releasing factor; Drinking in the dark; Ethanol; Extended amygdala; Ventral tegmental area;
Protein Kinase C Epsilon Activity in the Nucleus Accumbens and Central Nucleus of the Amygdala Mediates Binge Alcohol Consumption
Keywords: نوشیدن در تاریکی; Alcohol use disorders; Drinking in the dark; Glutamate; HDID-1 mice; Homer; Scheduled high alcohol consumption;
Research reportNeuropeptide Y response to alcohol is altered in nucleus accumbens of mice selectively bred for drinking to intoxication
Keywords: نوشیدن در تاریکی; Neuropeptide Y; Alcohol; Binge drinking; Drinking in the dark; Selective breeding; Behavioral genetics; Immunohistochemistry; Nucleus accumbens;
Role of interleukin-1 receptor signaling in the behavioral effects of ethanol and benzodiazepines
Keywords: نوشیدن در تاریکی; Flurazepam; Two-bottle choice; Drinking in the dark; Il1rn; Il1r1; Knockout mice; Kineret; Anakinra; Loss of righting reflex; Alcohol withdrawal; Conditioned taste aversion; IL-1ra; IL-1R1;
Behavioral disinhibition in mice bred for high drinking in the dark (HDID) and HS controls increases following ethanol
Keywords: نوشیدن در تاریکی; Inhibition; Go/No-Go; Drinking in the dark; Selected lines; Alcohol; Impulsivity;
Site-specific microinjection of Gaboxadol into the infralimbic cortex modulates ethanol intake in male C57BL/6J mice
Keywords: نوشیدن در تاریکی; Mouse; Drinking in the dark; Alcohol; GABA; Gaboxadol; Infralimbic cortex
Ethanol drinking in Withdrawal Seizure-Prone and -Resistant selected mouse lines
Keywords: نوشیدن در تاریکی; Ethanol withdrawal; Selective breeding; Ethanol preference; Drinking in the dark; Mouse; Genetics
Ethanol drinking-in-the-dark facilitates behavioral sensitization to ethanol in C57BL/6J, BALB/cByJ, but not in mu-opioid receptor deficient CXBK mice
Keywords: نوشیدن در تاریکی; Ethanol; Behavioral sensitization; Drinking in the dark; Naltrexone; Opioid system;
Limited access ethanol drinking in the dark in adolescent and adult mice
Keywords: نوشیدن در تاریکی; Adolescent; Ethanol; Drinking in the dark; Genetics; Mice; Development;
Increased consumption of ethanol and sugar water in mice lacking the dopamine D2 long receptor
Keywords: نوشیدن در تاریکی; Dopamine; D2 receptor; Knockout; Ethanol drinking; Alcoholism; Drinking in the dark