Fatty acid amide hydrolase inhibition enhances the anti-allodynic actions of endocannabinoids in a model of acute pain adapted for the mouse
Keywords: آندوکانابینوئید; fatty acid amide hydrolase; FAAH; endocannabinoid; allodynia; nociception; thermal injuryAEA, N-arachidonylethanolamide (anandamide); CB-1, cannabinoid-1 receptor; CB-2, cannabinoid-2 receptor; CDEX, 2-hydroxypropyl-β-cyclodextrin; FAAH, fatty acid amide