Keywords: تحریک-انقباض جفت شدن; EFS; electrical field stimulation; FOV; field of view; LTCC; L-type Ca2+ channel; NCX; Na+/Ca2+ exchanger; PSS; physiological salt solution; SR; sarcoplasmic reticulum; ROI; region of interest; RyR; ryanodine receptor; Pulmonary vein; Pulmonary vein cardi
مقالات ISI تحریک-انقباض جفت شدن (ترجمه نشده)
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Keywords: تحریک-انقباض جفت شدن; α-MHC; α-Myosin heavy chain; β-MHC; β-Myosin heavy chain; β-AS; β-adrenergic stimulation; CICR; Ca2+-induced Ca2+ release; CL; cell length; CS; citrate sintasa; CV; cell volume; CW; cell width; CD; contractile dysfunction; Ctrl; Control; CSA; cross-
Keywords: تحریک-انقباض جفت شدن; Heart failure; Myocardial biology; Excitation-contraction coupling;
Keywords: تحریک-انقباض جفت شدن; Testosterone; Myocardial infarction; Excitation-contraction coupling; Cardiac contraction; SERCA-2a;
Keywords: تحریک-انقباض جفت شدن; EMD; Excitation-contraction coupling; Series elastic component; Fatigue; Recovery;
Keywords: تحریک-انقباض جفت شدن; CoA; coenzyme A; TCA; tricarboxylic acid; [Ca2+]i; cytosolic free Ca2+; E-C; excitation-contraction; SR; sarcoplasmic reticulum; SERCA; SR Ca2+-ATPase; AMPK; adenosine monophosphate-activated protein kinase; MEF2; myocyte enhancer factor-2; PPAR; peroxiso
Keywords: تحریک-انقباض جفت شدن; [Ca2Â +]i; free cytosolic [Ca2Â +]; CSQ1; calsequestrin 1; COX1; cytochrome c oxidase subunit 1; CS; citrate synthase; Ctrl; control; DHPR; dihydropyridine receptor; EDL; extensor digitorum longus; Nit; nitrate; P/O; oxidative phosphorylation efficiency;
Keywords: تحریک-انقباض جفت شدن; Tetralogy of Fallot; Right ventricular function; Echocardiography; Speckle-tracking; Excitation-contraction coupling; Animal model; β-AR; β-adrenergic receptor; CMR; Cardiac magnetic resonance imaging; eEF; Efficient ejection fraction; FAC; Fractional a
Keywords: تحریک-انقباض جفت شدن; Optical mapping; Cardiac fibrillation; Excitation-contraction coupling; Image registration; Motion artifact; Motion tracking;
Keywords: تحریک-انقباض جفت شدن; AP; action potential; CICR; Ca2+-induced Ca2+-release; ECC; excitation-contraction coupling; LTCC; L-type Ca2+ channel; NCX; Na+/Ca2+-exchange; NCXrev; Na+/Ca2+-exchange in reverse mode; NIF; nifedipine; SERCA; sarcoendoplasmic reticulum Ca2+-ATPase; SR;
Keywords: تحریک-انقباض جفت شدن; Heart; Electrophysiology; Mechanics; Sinoatrial node; Atrium; Ventricle; Slow force response; AF; atrial fibrillation; AP; action potential; BR; beating rate; Ca2+; calcium; Clswell; swelling-activated chloride channel; ECC; excitation-contraction couplin
Keywords: تحریک-انقباض جفت شدن; AP; action potential; APD; action potential duration; [Ca2+]i; intracellular Ca2+ concentrations; CM; cardiomyopathy; CON; control; E-C; excitation-contraction; ICaL; L-type Ca current; IK1; inward rectifier current; IKr; rapid delayed rectifier current;
Keywords: تحریک-انقباض جفت شدن; Cardiomyocyte interaction; Excitation-contraction coupling; Cardiac mechano-electric coupling; Electro-mechanical mathematical model; Cardiac tissue; Mechano-electric feedback; 1D; One-dimensional; AP; Action potential; APD; Action potential duration; APD
Keywords: تحریک-انقباض جفت شدن; AKAP; A-kinase anchor protein; Ang II; angiotensin II; AP; action potential; APD; action potential duration; BH4; tetrahydrobiopterin; cAMP; cyclic adenosine monophosphate; CaM; calmodulin; CaMKII; Ca/calmodulin-dependent protein kinase II; CaMKIIδC; cyt
Keywords: تحریک-انقباض جفت شدن; Transverse friction massage; Electromechanical delay; Excitation-contraction coupling; Ultrafast ultrasonographyEC, excitation-contraction; EMD, electromechanical delay; EMG, electromyography; MTJ, myotendinous junction; QF, quadriceps femoris; RF, rectus
From excitation to intracellular Ca2+ movements in skeletal muscle: Basic aspects and related clinical disorders
Keywords: تحریک-انقباض جفت شدن; Excitation-contraction coupling; Ion channels; Myopathy; Intracellular Ca2+;
Cellular mechanisms of metabolic syndrome-related atrial decompensation in a rat model of HFpEF
Keywords: تحریک-انقباض جفت شدن; Heart Failure with preserved ejection fraction (HFpEF); Atrial function; Calcium (Ca) release; Excitation-contraction coupling; Angiotensin II;
Sorcin ablation plus β-adrenergic stimulation generate an arrhythmogenic substrate in mouse ventricular myocytes
Keywords: تحریک-انقباض جفت شدن; AP; action potential; BAPTA; 1,2-bis(o-aminophenoxy)ethane-N,N,Nâ²,Nâ²-tetraacetic acid; BVT; bidirectional ventricular tachycardia; CPVT; catecholaminergic polymorphic ventricular tachycardia; DAD; delayed afterdepolarization; EAD; early afterdepolariz
Targeting Calpain for Heart Failure Therapy
Keywords: تحریک-انقباض جفت شدن; calcium; calpain; excitation-contraction coupling; heart failure; T-tubules; CAPN1-OE; calpain-1 overexpressing; E-C coupling; excitation-contraction coupling; EF; ejection fraction; IP; intraperitoneally; ISO; isoproterenol; JP2; junctophilin-2; JP2-OE;
Loss of p21-activated kinase 1 (Pak1) promotes atrial arrhythmic activity
Keywords: تحریک-انقباض جفت شدن; Atrial fibrillation; Excitation-contraction coupling; NADPH oxidase-2; p21-activated kinase; Reactive oxygen species;
Transient activation of PKC results in long-lasting detrimental effects on systolic [Ca2+]i in cardiomyocytes by altering actin cytoskeletal dynamics and T-tubule integrity
Keywords: تحریک-انقباض جفت شدن; Cardiomyocytes; Protein kinase C; Actin cytoskeleton; T-tubules; Excitation-contraction coupling;
Contractile responses to endothelin-1 are regulated by PKC phosphorylation of cardiac myosin binding protein-C in rat ventricular myocytes
Keywords: تحریک-انقباض جفت شدن; ECC; Excitation-Contraction Coupling; CICR; Ca2+-induced Ca2+ release; ET-1; Endothelin-1; SR; Sarcoplasmic Reticulum; SERCA; SR/ER Ca2+ ATPase; NCX; Na+/Ca2+ exchanger; NHE; Na+/H+ exchanger; InsP3(R); Inositol 1,4,5-trisphosphate (receptor); PLC; Phosph
Enhanced effect of VEGF165 on L-type calcium currents in guinea-pig cardiac ventricular myocytes
Keywords: تحریک-انقباض جفت شدن; VEGF; vascular endothelial growth factor; LTCC; L-type calcium channel; VEGFR2; VEGF type-2 receptor; ICa,L; L-type calcium currents; AP; action potential; APD; action potential duration; EC coupling; excitation-contraction coupling; IV; current-voltage;
MG53 is dispensable for T-tubule maturation but critical for maintaining T-tubule integrity following cardiac stress
Keywords: تحریک-انقباض جفت شدن; Cardiomyocytes; MG53 (or TRIM72); T-tubules; Calcium; Sodium-calcium exchanger; Excitation-contraction coupling;
Effect of local cooling on excitation-contraction coupling in myasthenic muscle: Another mechanism of ice-pack test in myasthenia gravis
Keywords: تحریک-انقباض جفت شدن; Myasthenia gravis; Excitation-contraction coupling; Local cooling; Ice-pack test; Accelerometer; Masseter;
ArticleRole of FK506-binding protein in Ca2+ spark regulation
Keywords: تحریک-انقباض جفت شدن; Ca2+ spark; FK506-binding protein; Ryanodine receptor; Intracellular calcium; Excitation-contraction coupling;
β-Adrenergic induced SR Ca2 + leak is mediated by an Epac-NOS pathway
Keywords: تحریک-انقباض جفت شدن; Excitation-contraction coupling; Sarcoplasmic reticulum; Ryanodine receptor; Calcium calmodulin-dependent protein kinase; Epac; Nitric oxide synthase;
Two EF-hand motifs in ryanodine receptor calcium release channels contribute to isoform-specific regulation by calmodulin
Keywords: تحریک-انقباض جفت شدن; Excitation-contraction coupling; Skeletal muscle; Cardiac muscle; Ryanodine receptor; Ca2+ release channel; Calmodulin;
Membrane potential determines calcium alternans through modulation of SR Ca2Â + load and L-type Ca2Â + current
Keywords: تحریک-انقباض جفت شدن; Action potential; Alternans; Arrhythmia; Ca2Â + signaling; Excitation-contraction coupling; AP; action potential; APCaT_Large; AP recorded during a large amplitude alternans Ca2Â + transient; APCaT_Small; AP recorded during a small amplitude alternans Ca2
In vivo cardiac nano-imaging: A new technology for high-precision analyses of sarcomere dynamics in the heart
Keywords: تحریک-انقباض جفت شدن; Muscle; Contractility; Ventricular function; [Ca2+]i; Intracellular Ca2+ concentration; CaT; Calcium transient; EC coupling; Excitation-contraction coupling; ECG; Electrocardiogram; EMCCD; Electron-multiplying charge-coupled device; LV; Left ventricle; LV
Ca2+ handling abnormalities in early-onset muscle diseases: Novel concepts and perspectives
Keywords: تحریک-انقباض جفت شدن; Cav1.1; alfa 1 subunit of the dihydropyridine receptor; CCD; central core disease; CNM; centronuclear myopathy; DHPR; dihydropyridine receptor; ECC; excitation contraction coupling; ECCE; excitation coupled calcium entry; ER; endoplasmic reticulum; HDAC;
Impact of waterborne and trophic mercury exposures on cardiac function of two ecologically distinct Neotropical freshwater fish Brycon amazonicus and Hoplias malabaricus
Keywords: تحریک-انقباض جفت شدن; Fish; Mercury; ECG; Twitch force; Force frequency relationship; Excitation-contraction coupling; Sarcoplasmic reticulum;
Mechanisms involved in the in vitro contractile dysfunction induced by different concentrations of ferrous iron in the rat myocardium
Keywords: تحریک-انقباض جفت شدن; Iron poisoning; Iron intoxication; Skinned fiber; Excitation-contraction coupling; Papillary muscle;
JPH-2 interacts with Cai-handling proteins and ion channels in dyads: Contribution to premature ventricular contraction-induced cardiomyopathy
Keywords: تحریک-انقباض جفت شدن; Ab; antibody; BIN-1; bridging integrator-1; Cavβ2; voltage-gated Ca channel β subunit 2; CICR; Ca-induced Ca release; CON; control; CSQ2; calsequestrin 2; ICaL; L-type Ca channel current; IKs; slow delayed rectifier current; IP; immunoprecipitate; JPH-2
MDIMP, a novel cardiac Ca2+ channel blocker with atrial selectivity
Keywords: تحریک-انقباض جفت شدن; Ion channels; Intracellular Ca2+; Cardiac myocyte; Excitation-contraction coupling; Ca2+-induced Ca2+ release
Role of cardiac troponin I carboxy terminal mobile domain and linker sequence in regulating cardiac contraction
Keywords: تحریک-انقباض جفت شدن; Heart; Thin filament; In vitro motility assay; Unloaded filament sliding; Calcium; Excitation-contraction coupling;
In vitro myogenesis induced by human recombinant elastin-like proteins
Keywords: تحریک-انقباض جفت شدن; Elastin-like polypeptides; Biomimetic materials; Cell adhesion; Skeletal muscle regeneration; Excitation-contraction coupling; Intracellular calcium;
Genetic modulation of the SERCA activity does not affect the Ca2+ leak from the cardiac sarcoplasmic reticulum
Keywords: تحریک-انقباض جفت شدن; Cardiac muscle; Excitation-contraction coupling; Aequorin; Ca2+ transient; Transgenic mouse;
Novel excitation-contraction uncoupled RYR1 mutations in patients with central core disease
Keywords: تحریک-انقباض جفت شدن; Central core disease; Congenital myopathies; Ryanodine receptor type 1; Calcium transport; Excitation-contraction coupling;
Mitochondria-mediated cardioprotection by trimetazidine in rabbit heart failure
Keywords: تحریک-انقباض جفت شدن; ÎΨm; mitochondrial membrane potential; 4-BA; 4-bromotiglic acid; [Ca2 +]i; cytosolic calcium concentration; [Ca2 +]em; extramitochondrial free calcium; CAT; carboxyatractyloside; ecc; excitation-contraction coupling; ETC; electron transport chain; FAO
Carbon monoxide exposure in the urban environment: An insidious foe for the heart?
Keywords: تحریک-انقباض جفت شدن; Excitation-contraction coupling; ECG; Arrhythmia; Cardiac remodelling; Ca2+ handling;
Hypoxia and HIF-1 suppress SERCA2a expression in embryonic cardiac myocytes through two interdependent hypoxia response elements
Keywords: تحریک-انقباض جفت شدن; Calcium cycling; Excitation-contraction coupling; Sarcoplasmic reticulum; Gene expression; Gene promoter;
Excitation–contraction coupling in human heart failure examined by action potential clamp in rat cardiac myocytes
Keywords: تحریک-انقباض جفت شدن; Action potential; Heart failure; Human; Animal model; Ca2+; Excitation-contraction coupling
Cardiac action potential duration and calcium regulation in males and females
Keywords: تحریک-انقباض جفت شدن; L-type Ca2+ current; Action potential; Calcium; Sarcoplasmic reticulum Ca2+ content; Excitation-contraction coupling; Male; Female;
Mitochondrial Ca2+ uptake: Tortoise or hare?
Keywords: تحریک-انقباض جفت شدن; Calcium uniporter; Sodium–calcium exchange; Mitochondrial inner membrane; Ca2+ transients; Energy metabolism; Cellular energetics; Excitation-contraction coupling
Architecture moléculaire du réticulum sarcoplasmique et son rôle dans le couplage excitation-contraction
Keywords: تحریک-انقباض جفت شدن; Sarcoplasmic reticulum; Excitation-contraction coupling; Ryanodine receptor; Dihydropyridine receptor; Calcium; Réticulum sarcoplasmique; Couplage excitation-contraction; Récepteur à la ryanodine; Récepteur à la dihydropyridine; Calcium;
Alternative splicing of RyR1 alters the efficacy of skeletal EC coupling
Keywords: تحریک-انقباض جفت شدن; Skeletal ryanodine receptor; Variably spliced residues; Excitation-contraction coupling; Myotonic dystrophy; Development; Nuclear magnetic resonance; Structure
Nitric oxide signaling and the regulation of myocardial function
Keywords: تحریک-انقباض جفت شدن; NOS; Peroxynitrite; cGMP; Excitation-contraction coupling; l-type Ca2+ channel; Phospholamban; Ryanodine receptor
Ca2+ sparks and cellular distribution of ryanodine receptors in developing cardiomyocytes from rat
Keywords: تحریک-انقباض جفت شدن; Newborn; Ryanodine receptor; Ca2+ sparks; Excitation-contraction coupling; Transverse tubules
Functional and developmental properties of human embryonic stem cells–derived cardiomyocytes
Keywords: تحریک-انقباض جفت شدن; Human embryonic stem cells–derived cardiomyocytes; Excitation-contraction coupling; [Ca2+]i transients and contractions; Sarcoplasmic reticulum Ca2+ release