
Prenatal alcohol exposure potentiates chronic neuropathic pain, spinal glial and immune cell activation and alters sciatic nerve and DRG cytokine levels
Keywords: اختلال طیف الکل جنین; Neuropathic pain; Chronic constriction injury; β-Integrin adhesion molecules; Prenatal alcohol exposure; Fetal alcohol spectrum disorder; Inflammatory cytokines; Spinal glia; Astrocytes; Microglia; Sciatic nerve; Neuroimmune interactions;