
A critical appraisal of the role of intracellular Ca2+-signaling pathways in Kawasaki disease
Keywords: [Ca2+]cyt; free cytosolic Ca2+ concentration; CRAC; Ca2+-release activated Ca2+ entry; EBV; Epstein-Barr virus; ER; endoplasmic reticulum; IP3; inositol 1,4,5-trisphosphate; IP4; inositol 1,3,4,5-tetrakisphosphate; IP3R; IP3 receptor; KD; Kawasaki disease