
LXR agonism improves TNF-α-induced endothelial dysfunction in the absence of its cholesterol-modulating effects
Keywords: LXR agonist; TNF-α; Endothelial dysfunction; Nitric oxide; HUVEC; human umbilical vein cells; LXR; Liver X receptor; NO; nitric oxide; eNOS; endothelial nitric oxide synthase; NOX; NAD(P)H oxidase; O2â¢; superoxide; ROS; reactive oxygen species; RXR; re