Prolonged treatment with mevalonolactone induces oxidative stress response with reactive oxygen species production, mitochondrial depolarization and inflammation in human glioblastoma U-87 MG cells
Keywords: اسیدوریا میوالنیک; Mevalonate kinase deficiency; Mevalonolactone; Human glioblastoma U-87â¯MG cells; Oxidative stress; Mitochondrial damage; Inflammation; ACTB; actin β gene; H2DCFDA; 2â²,7â²-dichlorofluorescein diacetate; HemeOX; heme oxygenase gene; HIDS; Hyper IgD Sy