Modelling the pathogenesis of Myotonic Dystrophy type 1 cardiac phenotype through human iPSC-derived cardiomyocytes
Keywords: Myotonic Dystrophy type 1 (DM1); Human induced pluripotent stem cells; Cardiomyocytes; Electrophysiological analysis; Antiarrhytmic drugs; ACTN2; sarcomeric alpha actinin; AFM; atomic force microscopy; AP; action potential; APD; action potential duration;