Keywords: Pain; Neurotoxicant; Nociceptor; Gulf War Illness; Deactivation; Nav1.8;
مقالات ISI (ترجمه نشده)
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Involvement of TRPV1 and TDAG8 in Pruriception Associated with Noxious Acidosis
Keywords: ASIC; acid-sensing ion channel; CP48/80; compound 40/80; DRG; dorsal root ganglion; ET; endothelin; GPCR; G-protein-coupled receptor; HEPES; 4-(2-hydroxyethyl)-1-piperazineethanesulfonic acid; HT; hydroxytryptamine; IB4; isolectin B4; IRES; internal rib
Oxidation differentially modulates the recombinant voltage-gated Na+ channel α-subunits Nav1.7 and Nav1.8
Keywords: TTX; tetrodotoxin; Nav; voltage-gated sodium channel; TRP; transient receptor potential; HEK; human embryonic kidney; EGFP; green fluorescent protein; DRG; dorsal root ganglion; Nociception; Pain; Oxidative stress; Sodium channel; Nav1.7; Nav1.8;
Methylglyoxal mediates streptozotocin-induced diabetic neuropathic pain via activation of the peripheral TRPA1 and Nav1.8 channels
Keywords: Methylglyoxal; Diabetic neuropathic pain; TRPA1; Nav1.8; Methylglyoxal scavengers; AGE; advanced glycation end-product; TRPA1; transient receptor potential channel ankyrin 1; AMPK; AMP-activated protein kinase; DMSO; dimethyl sulfoxide; PEG400; polyethyle
A novel substituted aminoquinoline selectively targets voltage-sensitive sodium channel isoforms and NMDA receptor subtypes and alleviates chronic inflammatory and neuropathic pain
Keywords: Chronic pain; NMDA; GluN2B; Nav1.8; Nav1.7; Substituted aminoquinoline
Intrathecal administration of rapamycin inhibits the phosphorylation of DRG Nav1.8 and attenuates STZ-induced painful diabetic neuropathy in rats
Keywords: STZ; streptozotocin; PDN; painful diabetic neuropathy; DRG; dorsal root ganglion; mTOR; mammalian target of rapamycin; S6K; ribosomal protein S6 kinase; 4E-BP; eukaryotic initiation factor 4E (eIF4E) binding protein; IR; insulin receptor; IRS-1; insulin r
Wound-healing growth factor, basic FGF, induces Erk1/2-dependent mechanical hyperalgesia
Keywords: Pain; Nociception; Peripheral sensory neuron; Sensitization signaling; Inflammatory pain; Wound healing; Quantitative automated microscopy; bFGF; FGF-2; FGF-β; MAP kinase; Erk1/2; NaV1.8; Voltage gated sodium channels; Randall Selitto;
The scorpion toxin Amm VIII induces pain hypersensitivity through gain-of-function of TTX-sensitive Na+ channels
Keywords: Voltage-gated sodium channels; Nav1.7; Nav1.8; Nav1.9; Excitability; Sensory neurons; Allodynia; Hyperalgesia; Pain; Scorpion toxins;
Protein Kinase B/Akt Is Required for Complete Freund's Adjuvant-Induced Upregulation of Nav1.7 and Nav1.8 in Primary Sensory Neurons
Keywords: Nav1.7; Nav1.8; Akt; dorsal root ganglion; inflammatory pain
Persistent Na+ and K+ channel dysfunctions after chronic exposure to insecticides and pyridostigmine bromide
Keywords: Pain; Neurotoxicant; Nociceptor; Gulf War; Nav1.8; Kv7
Expression of Transient Receptor Potential Ankyrin 1 in Human Dental Pulp
Keywords: Cold hypersensitivity; dental pulp; immunohistochemistry; inflammation; Nav1.8; nociception; TRPA1
Up-regulation of low-threshold tetrodotoxin-resistant Na+ current via activation of a cyclic AMP/protein kinase A pathway in nociceptor-like rat dorsal root ganglion cells
Keywords: NaV1.9; NaV1.8; A-803467; sensory neuron; hyperalgesia; inflammationAC, adenylyl cyclase; A-D/D-A, analog to digital/digital to analog; cAMP, cyclic AMP; DRG, dorsal root ganglion; EGTA, ethylene glycol tetraacetic acid; GTP, guanosine triphosphate; HEPES
Subtype-selective Nav1.8 sodium channel blockers: Identification of potent, orally active nicotinamide derivatives
Keywords: Sodium channel blocker; Nav1.8; PN3; Pain
Serotonin upregulates low- and high-threshold tetrodotoxin-resistant sodium channels in the same subpopulation of rat nociceptors
Keywords: NaV1.8; NaV1.9; hyperalgesia; sensory neuron; dorsal root ganglion; A-D/D-A; analog to digital/digital to analog; CP; conditioning potential; DRG; dorsal root ganglion; EGTA; ethylene glycol tetraacetic acid; GTP; guanosine triphosphate; HEPES; 4-(2-hydro
Functional tetrodotoxin-resistant Na+ channels are expressed presynaptically in rat dorsal root ganglia neurons
Keywords: Nav1.8; Nav1.9; SBFI; tetrodotoxin; synaptic transmission; sensory synapseara-C, cytosine-A-D-arabinofuranoside; [Ca2+]i, intracellular Ca2+ concentration; DRG, dorsal root ganglia; EGFP, enhanced green fluorescent protein; EPSC, excitatory postsynaptic c
Quantification of Neural Protein in Extirpated Tooth Pulp
Keywords: NaV1.8; nociceptor; pain; primary afferent
Discovery of potent furan piperazine sodium channel blockers for treatment of neuropathic pain
Keywords: Furan piperazine; Sodium channels; Voltage-gated; Blocker; Nav1.8; PN3; Pain
Neurotrophin-3 significantly reduces sodium channel expression linked to neuropathic pain states
Keywords: Nav1.8; Nav1.9; DRG; Sciatic nerve; CCI; Nociceptor; Nerve growth factor;
Burning mouth syndrome as a trigeminal small fibre neuropathy: Increased heat and capsaicin receptor TRPV1 in nerve fibres correlates with pain score
Keywords: TRPV1; NGF; Nav1.8; Burning mouth syndrome
Comparison of the effects of four Na+ channel analgesics on TTX-resistant Na+ currents in rat sensory neurons and recombinant Nav1.2 channels
Keywords: Na+ channel; Nav1.2; Nav1.8; Mexiletine; Lidocaine; Benzocaine; Ambroxol;
The neuroleptic drug, fluphenazine, blocks neuronal voltage-gated sodium channels
Keywords: Sodium channel; Fluphenazine; Neuroleptic; NaV1.8; Electrophysiology; Neuron
Analysis of the variation in use-dependent inactivation of high-threshold tetrodotoxin-resistant sodium currents recorded from rat sensory neurons
Keywords: dorsal root ganglia; nociceptor; NaV1.8; TTX-resistant Na+ current; firing rate adaptation; sodium current inactivationANOVA, analysis of variance; DRG, dorsal root ganglia; EGTA, ethylene glycol-bis (β-aminoethylether)-N,N,N′,N′-tetraacetic acid; f, fitt
Localization of the Nav1.8 sodium channel isoform at nodes of Ranvier in normal human radicular tooth pulp
Keywords: Nav1.8; Sodium channels; Human; Tooth pulp; Nodes of Ranvier; caspr;
Ambroxol, a Nav1.8-preferring Na+ channel blocker, effectively suppresses pain symptoms in animal models of chronic, neuropathic and inflammatory pain
Keywords: Ambroxol; Na+ channel; Nav1.8; Neuropathic pain; Inflammatory pain; Chronic pain;