
Keywords: بیماری های نوروژنیک; Extracellular alpha-synuclein; Oxidative stress; “Prion-like” protein transmission; Nitric oxide; Mitochondria dysfunction; Neurodegenerative diseases; OH; hydroxyl radicals; AD; Alzheimer's disease; ASN; α-synuclein; Aβ; amyloid β; CaN; calcineu