
Reduction of thalamic and cortical Ih by deletion of TRIP8b produces a mouse model of human absence epilepsy
Keywords: تخلیه موج سنبله; HCN; hyperpolarization-activated cyclic-nucleotide-gated; RTN; reticular thalamic nucleus; SWD; spike-wave discharge; TC; thalamocortical; TRIP8b; tetratricopeptide-containing Rab8b-interacting protein; TRIP8b; HCN channels; Ih; Absence epilepsy; Neocor