Smooth muscle cell-driven vascular diseases and molecular mechanisms of VSMC plasticity
Keywords: عامل پیچیده سه گانه; Vascular smooth muscle; Vasoproliferative disease; Vascular smooth muscle cell; Phenotype switching; Signalling pathways; AJ; adherens junction; ApoE; apolipoprotein E; BMP; bone morphogenetic protein; CAD; coronary artery diseases; CADASIL; cerebral auto