Shock wave induced damage in kidney tissue

The material model combines shear induced finite plasticity with irreversible volumetric expansion as induced, e.g., by cavitating bubbles. Dynamic effects like micro-inertia and rate sensitivity are included. The time-discretized porous-viscoplastic constitutive updates are described in a fully variational manner. A finite element analysis localizes the damage in the human kidney in good agreement to clinical and experimental studies.