Cellular stress and AMPK activation as a common mechanism of action linking the effects of metformin and diverse compounds that alleviate accelerated aging defects in Hutchinson-Gilford progeria syndrome
Keywords: جابجایی جایگزین; AMP-activated protein kinase (AMPK); Hutchinson-Gilford progeria syndrome (HGPS); Oocyte; HIV-1 latency; Rapamycin; Metformin; Retinoic acid; Methylene blue; Sulforaphane; Oltipraz; Vitamin D; MG132; Acrosome; Alternative splicing; ULK1; Autophagy; PGC-1Î