Keywords: PM2.5; BEAS-2B; Epithelial-mesenchymal transition; Cell invasion;
مقالات ISI (ترجمه نشده)
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Keywords: Acute inhalation toxicity; In vitro; In silico; Ex vivo; Quantitative structure-activity relationships (QSAR); Adverse outcome pathway; Aggregate exposure pathway; Dosimetry; Integrated approach to testing and assessment (IATA); Risk assessment; ADME; abs
Keywords: Dp; Dermatophagoides peronyssinus; Df; Dermatophagoides farinae; HDM; house dust mite; Der p1; group 1 allergen of D. peronyssinus; Der p2; group 2 allergen of D. peronyssinus; Der p3; group 3 allergen of D. peronyssinus; Der p7; group 7 allergen of D. pe
Keywords: PM2.5; BEAS-2B; Aryl hydrocarbon receptor (AhR); Reactive oxygen species (ROS); Nuclear factor κB (NF-κB);
Keywords: BEAS-2B; Copper; Cytotoxicity; Genotoxicity; Pseudomonas aeruginosa; Viable but non culturable (VBNC);
Keywords: As; Arsenic; As(III); Trivalent arsenic; APC; Adenomatous polyposis coli; BEAS-2B; Human bronchial epithelial cells; Cd; Cadmium; Cr; Chromate; C57B1/6; Mouse embryonic stem cell; ECM; Extracellular matrix; GILA; Grow in low attachment; IARC; Internationa
Keywords: A549; human lung adenocarcinoma; ANOVA; analysis of variance; b.wt; body weight; BEAS-2B; human bronchial epithelial cells; CAs; chromosomal aberrations; CAT; catalase; CNTs; carbon nanotubes; Cu; copper; DMSO; dimethylsulfoxide; DNA; deoxyribonucleic aci
Keywords: FLIM; fluorescence lifetime imaging microscopy; HE; haematoxylin and eosin; NADH; reduced nicotinamide adenine dinucleotide; FAD; flavin adenine dinucleotide; TCSPC; time-correlated single photon counting; BEAS-2B; human bronchial epithelial cells; Autofl
Keywords: Biodiesel; BEAS-2B; Cytotoxicity; Cytokines; Cell proliferation; Metal; Particles; PAH;
Keywords: Tungsten; Cancer; Beas-2B; RNA-Seq; In vitro; Nude mice
Keywords: Building-construction; Particulate matter; Risk assessment; Cytotoxicity; BEAS-2B; RAW 264.7; High-content-screening; MATLAB modeling
Keywords: BEAS-2B; Silica nanoparticles; Cytokine responses; Cytokine gene arrays; Size and surface area; Exposure conditions;
Keywords: Cobalt oxide; Cerium dioxide; Nanoparticles; BEAS-2B; A549; Transcriptomics
miRNA-mediated apoptosis activation through TMEM 48 inhibition in A549 cell line
Keywords: Lung cancer; TMEM48; miRNA; miR-421; A549; Beas-2B;
Fetal bovine serum induces sustained, but reversible, epithelial-mesenchymal transition in the BEAS-2B cell line
Keywords: BEAS-2B; Epithelial-mesenchymal transition; Respiratory; Epithelial; Cell culture; TGF;
Identification of 5-hydroxymethylfurfural in cigarette smoke extract as a new substrate metabolically activated by human cytochrome P450 2A13
Keywords: CYP; cytochrome P450; CYP2A13; cytochrome P450 2A13; CYP2A5; cytochrome P450 2A5; 8-MOP; 8-methoxypsoralen; BEAS-2B; immortalized human bronchial epithelial cells; B-2A13; BEAS-2B cells stably expressing CYP2A13; CSE; cigarette smoke extract; TSNA; tobacc
Gamma-irradiation-killed Streptococcus pneumoniae potently induces the expression of IL-6 and IL-8 in human bronchial epithelial cells
Keywords: Streptococcus pneumoniae; Gamma-irradiation; BEAS-2B; Vaccine; Innate immunity;
Calpeptin attenuates cigarette smoke-induced pulmonary inflammation via suppressing calpain/IκBα signaling in mice and BEAS-2B cells
Keywords: CS; cigarette smoke; COPD; chronic obstructive pulmonary disease; CSE; cigarette smoke extract; PS; physiological saline; EPI or EPII; type I or II alveolar epithelial cells; NF-κB; nuclear factor-κB; DMSO; dimethylsulfoxide; NC; normal control; CS1-3;
Novel nuclear translocation of inositol polyphosphate 4-phosphatase is associated with cell cycle, proliferation and survival
Keywords: INPP4A; inositol polyphosphate 4 phosphatase type I enzyme; LMB; leptomycin B; PtdIns(P); phosphatidyl inositol phosphate; Ins(1,4,5)P3; Inositol 1,4,5 tris-phosphate; PDK-1; phosphoinositide-dependent kinase-1; mTORC2; mammalian target of rapamycin compl
Evaluation of whole cigarette smoke induced oxidative stress in A549 and BEAS-2B cells
Keywords: Whole cigarette smoke; Air-liquid interface; A549; BEAS-2B; Oxidative stress;
Cigarette Smoke-Induced Hypermethylation of the GCLC Gene Is Associated With COPD
Keywords: antioxidants; COPD; smoking; 5-aza; 5-aza-2ʹ-deoxycytidine; BEAS-2B; human bronchial epithelial; CPG; C-phosphate-G; CS-COPD; current smoker with COPD; CSE; cigarette-smoke extract; CS-NS; current smoker with normal lung function; ES-COPD; exsmoker with
House dust mite-induced asthma causes oxidative damage and DNA double-strand breaks in the lungs
Keywords: House dust mite; asthma; oxidative stress; DNA double-strand breaks; BEAS-2B; cell death; DNA repair; NU7441; AHR; Airway hyperresponsiveness; ATM; Ataxia-telangiectasia mutated; BAL; Bronchoalveolar lavage; CC10; Club cell 10Â kDa protein; DCFH-DA; 2,7-D
Organic extracts from African dust storms stimulate oxidative stress and induce inflammatory responses in human lung cells through Nrf2 but not NF-κB
Keywords: ADE; African dust events; Non-ADE; non African dust events; BEAS-2B; human bronchial epithelial cells; ROS; reactive oxygen species; DF; deferoxamine mesylate; NAC; N-acetyl-l-cysteine; GSTP1; glutathione-S-transferase; HMXO1; hemeoxygenase 1 mRNA; Nrf2;
Cadmium induces cytotoxicity in human bronchial epithelial cells through upregulation of eIF5A1 and NF-kappaB
Keywords: Cadmium; BEAS-2B; eIF5A1; NF-kappaB; Cell death;
Role of Frizzled6 in the molecular mechanism of beta-carotene action in the lung
Keywords: BC; β-carotene; BCMO1; beta-carotene 15,15â²-monooxygenase 1; Fzd6; frizzled homologue 6; Cthrc1; collagen triple helix repeat containing 1; GO; Gene Ontology; DAVID; Database for Annotation Visualization and Integrated Discovery; DO; Disease Ontology;
Particle pollution in Rio de Janeiro, Brazil: Increase and decrease of pro-inflammatory cytokines IL-6 and IL-8 in human lung cells
Keywords: Particles; BEAS-2B; Industry; Cytokines; Decrease; Metals;
Mutagenicity and clastogenicity of native airborne particulate matter samples collected under industrial, urban or rural influence
Keywords: Air pollution; Particulate matter; BEAS-2B; Emission sources; Seasonal variations;
Silica nanoparticles induce cytokine responses in lung epithelial cells through activation of a p38/TACE/TGF-α/EGFR-pathway and NF-κΒ signalling
Keywords: BEAS-2B; Particles; Intracellular signalling; Cytokines
Nicotine mediates oxidative stress and apoptosis through cross talk between NOX1 and Bcl-2 in lung epithelial cells
Keywords: AEC; alveolar epithelial cell; Bis I; bisindolylmaleimide I; BEAS-2B; immortalized human bronchial epithelial cells; DHE; dihydroethidium; DMSO; dimethyl sulfoxide; ER; endoplasmic reticulum; GKT136901; NOX1/NOX4 dual inhibitor; mKRB; modified Krebs-Rin
Analysis of nicotine-induced DNA damage in cells of the human respiratory tract
Keywords: Genotoxicity; Nicotine; Human study; Nasal mucosa; BEAS-2B; Apoptosis
Neutrophils augment LPS-mediated pro-inflammatory signaling in human lung epithelial cells
Keywords: BEAS-2B; human bronchial epithelial cultured cell line; CPH; 1-hydroxy-3-carboxy-2,2,5,5-tetramethylpyrrolidine; DUOX1/2; dual oxidases 1 and 2; EPR; electron paramagnetic resonance; ERK1/2; extracellular signal-regulated kinases 1/2; H2O2; hydrogen perox
p52–Bcl3 complex promotes cyclin D1 expression in BEAS-2B cells in response to low concentration arsenite
Keywords: NF-κB; Bcl3; Cyclin D1; Arsenite; BEAS-2B; Lung cancer
Titanium dioxide nanoparticles cause apoptosis in BEAS-2B cells through the caspase 8/t-Bid-independent mitochondrial pathway
Keywords: Titanium dioxide nanoparticles; Apoptosis; Caspases; BEAS-2B
Genome-wide analysis of BEAS-2B cells exposed to trivalent arsenicals and dimethylthioarsinic acid
Keywords: Arsenic; Oxidative stress; BEAS-2B; Lungs; Genomics; DEGs (differentially expressed genes)
Wood dusts induce the production of reactive oxygen species and caspase-3 activity in human bronchial epithelial cells
Keywords: Wood dust; Reactive oxygen species; Caspase-3; Apoptosis; Bronchial epithelial cell; BEAS-2B
Inflammation in methotrexate-induced pulmonary toxicity occurs via the p38 MAPK pathway
Keywords: Inflammation; Interleukin (IL)-8; Methotrexate (MTX); p38 Mitogen-activated protein kinase (MAPK); Pulmonary toxicity; BEAS-2B
Proteomic analysis of proteins associated with tt-DDE induced toxicity in BEAS-2B cells
Keywords: tt-DDE; BEAS-2B; Proteomics; RP-nano-HPLC-ESI-MS/MS; 2DE;
Effects of cell type and culture media on Interleukin-6 secretion in response to environmental particles
Keywords: Particulate matter; Environmental dust; Soil; Coal; Lung; Epithelial cell; Human; Interleukin-6; BEAS-2B; A549; RAW264.7; Macrophage
Influence of platinum, palladium and rhodium as compared with cadmium, nickel and chromium on cell viability and oxidative stress in human bronchial epithelial cells
Keywords: Platinum group elements; Lung epithelial cells; BEAS-2B; MTT assay; DCF assay; ROS
Comparative acute and combinative toxicity of aflatoxin B1 and fumonisin B1 in animals and human cells
Keywords: Aflatoxin B1; Fumonisin B1; Rats; Mosquitofish; HepG2; BEAS-2B; Acute; Combinative; ToxicityAFs, aflatoxins; AFB1, aflatoxin B1; BEAS-2B, immortalized human bronchial epithelial cells; FNs, fumonisins; FB1, fumonisin B1; HepG2, immortalized human hepatoma
Beta-carotene uptake and metabolism in human lung bronchial epithelial cultured cells depending on delivery vehicle
Keywords: Beta-carotene; Lung cancer; BEAS-2B; Dimethylsulfoxide; Beadlet;
Expression of B7-H1 and B7-DC on the airway epithelium is enhanced by double-stranded RNA
Keywords: Innate immunity; Double-stranded RNA; IFN-γ; IL-13; Programmed death-1; BEAS-2B; T cell clone; Glucocorticoid;
Expression of CXC Chemokine Receptors 1 and 2 in Human Bronchial Epithelial Cells
Keywords: BEAS-2B; bronchial epithelium; chemokine receptor; CXC chemokine receptor 1; CXC chemokine receptor 2; flow cytometry; real-time amplification; CXCR1 = CXC chemokine receptor 1; CXCR2 = CXC chemokine receptor 2; FACS = fluorescence activated cell sorter;