Fragment 31–35 of β-amyloid peptide induces neurodegeneration in rat cerebellar granule cells via bax gene expression and caspase-3 activation: A crucial role for the redox state of methionine-35 residue
Keywords: سلول های گرانول مخچه; Alzheimer's disease; Amyloid β-peptide; AβP(31–35) fragment; Methionine; Cerebellar granule cells; Apoptosis