Mitigation of augmented extrasynaptic NMDAR signaling and apoptosis in cortico-striatal co-cultures from Huntington's disease mice
Keywords: فرهنگ سازی; NMDA receptor; GluN2A; GluN2B; Huntington's disease; Neurodegeneration; Striatal medium spiny neuron; Excitotoxicity; Glutamate; Extrasynaptic; Corticostriatal; Coculture; pCREB; Microfluidic isolation chamber; Electrophysiology; YAC128; Striatum;